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Engineered Epidemic

Jay Van Liere
e.coli.jpg

Positive forensic evidence has surfaced indicating that the e. coli superstrain that recently ravaged Europe, was created by humans. 

Science has proved that the bacteria is resistant to 8 different types of antibiotics and possesses DNA sequences from plague bacteria, a combination utterly unprecedented in nature, which has many authorities theorizing it was created in a lab and either released purposefully (with, perhaps, the intent to control global food and health), or accidentally into the food supply.

This distinct e. coli variation is part of the 0104 strain, and these strains are essentially never (under natural circumstances) resistant to antibiotics.  To acquire this capacity they must be repetitively exposed to antibiotics in order to incite the “mutation pressure” that nudges them toward complete drug immunity.  To unveil the origins of such a strain, science basically reverse-engineers the genetic code of the e. coli to determine which antibiotics it was exposed to during its development.  Decoding the genetic makeup of the 0104 strain, scientists at Germany's Robert Koch Institute discovered it to be immune to all the following classes and combinations of antibiotics: 1) penicillins 2) tetracycline
3) nalidixic acid 4) trimethoprim-sulfamethoxazol 5) cephalosporins 6) amoxicillin / clavulanic acid
7) piperacillin-sulbactam 8) piperacillin-tazobactam.                        

Bacterial resistance to a single antibiotic is common; but the creation of a strain of e. coli that's immune to 8 different classes of antibiotics defies the laws of genetic permutation and combination in nature.


Also, antibiotics are not used in vegetables, so the bacteria most likely was not exposed to even one form, much less 8.  If one chooses to believe that this superbug evolved separate of human intervention in the wild, this would be an even more alarming scenario: for it would mean this octobiotic superstrain developed randomly of its own accord, and that other similar superbugs could simply appear anywhere, anytime, without cause.  This would then simply be Mother Nature creating a 12 Monkeys scenario – far scarier, yet quite impossible in the opinion of most of the scientific community.

The real scheme at work here appears to be “problem, reaction, solution,” as pointed out by Natural News editor, Mike Adams.  Initially, create a problem (infections caused by ); then wait for the expected reactionism from the public (fear-based outcries and willingness to accept totalitarianism); then bring forth a solution (complete control over the global food supply and the outlawing of healthy raw foods, herbs, and supplements).  Ironically, the outbreak occurred just after the European Union's ban on herbal medicine and supplements.  Worldwide, the trend to control global food supply, nutrients, and any semblance of self-remedy is now being realized by much of humanity. 

By riding on the fear of e. coli outbreaks in the U.S., the FDA was able to strangle farm freedom domestically.  Here it relied on this same psychological phenomenon when vying for its fairly recent "Food Safety Modernization Act", which basically outlaws small organic family farms unless they pander to FDA regulators.  America is now seeing violent tactical government raids on organic grocery stores and cooperatives; like the recent narcotic-style raid and illegal seizure of properties at Rawsome Foods in Venice, CA

If they aren’t being created by small organic farms and raw food spots, what could be the cause behind the existence of such super pathogens?  Suspicions hover around the same bad guys in-depth investigators keep unmasking again and again; such as: megalithic and destructive agricultural giants like Monsanto, big pharma and the Western medicine complex, and bioweaponry facilities – summarily, part of a collective that is desperately trying to become the new world government.  They are trying to demonize organic, raw food and alternative medicines, to bring about their own global control and regulation of the food supply chain and medical treatment.  In the United States, Obama has recently appointed Michael Taylor, Monsanto's VP and chief lobbyist, as senior adviser to the commissioner of the FDA.  Formerly, Taylor was policy chief of the FDA when GMO's were allowed into the US food supply without undergoing a single test to determine their safety.  On the bright side....the bad guys are not getting away with it unnoticed these days.

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Comments

There's a much simpler

Submitted by seachanged on Sun, 09/25/2011 - 02:14.
There's a much simpler explanation. Someone who worked on those vegetables was in a hospital recently, and got an infection of an antibiotic-resistant strain of E. coli. Then they contaminated their hands with their fecal matter, and contaminated the food with it.

The more complicated scientific explanation

Submitted by tentontoby on Fri, 09/30/2011 - 10:24.
Prof. Helge Karch http://www.seismoblog.de/2011/06/11/ehec-ausbruchsstamm-prof-karch-vermu... virtually ruled out that the infection takes place only through contact from person to person. Karch: "It must rather be assumed that other infectious agents, for example, have been contaminated by human feces, have a meaning. These kinds of infectious agents must of course be found." and a response from a fb friend: "These bugs use what is known as the oral-fecal route of transmission, which is just as nasty as it sounds. The human to human oral-fecal route now seems to be established for this, but there's a good chance there will turn out to be animal reservoirs as well. It'll take awhile to sort out. Incidentally, it's now come to light that there have been sporadically at least four or five previous cases or small clusters of Serotype O104:H4 causing human disease - Germany in 2001, France 2004, Korea 2005, Republic of Georgia in 2009, Finland in 2010.... It's also come clear over the last couple of days that it's a fairly typical enteroaggregative E. coli strain that has picked up a bacteriophage (a bacterial virus) which encodes Stx/VT (shigatoxin / Verotoxin) - well known and widely distributed virulence factors more usually associated with "bad hamburger" type of E. coli (O157:H7 is the classic example). Thankfully, this particular outbreak seems to be winding down. Bacterial genetics, genomics, and pathogenesis are areas where I have very considerable professional expertise. And I'm quite comfortable in stating that one needn't invoke intentional engineering of a biological weapon to very easily explain the emergence of this strain. In fact, I see things like this as more of an absolutely inevitable outcome of the horrible practices of large scale agribusiness and a culture where patients demand a prescription antibiotic for every sniffle. You raise an interesting point about anti-bacterial propaganda..it's just gone too far. For instance, it's pretty amazing how in the last ten years, hand sanitizers are suddenly everywhere... even in the fricking grocery store so you can wipe off the handle of your shopping cart. It's absurd. I believe that the modern trend of raising children in an antiseptic bubble is horribly misguided, and likely contributes significantly to the increasing prevalence of everything from childhood asthma to food allergies. It seems pretty clear to me that developing a healthy immune system requires that children do things like play in the dirt and have naptime snuggles with the family's dog. Speaking now as a bacterial geneticist, there's a few things that are important to understand about this outbreak. The first is the fact that, while this bug indeed represents a novel combination of virulence factors, pretty much all the constituent factors have been floating around in natural populations for years. The next thing to understand is that while it seemingly has an impressive spectrum of multidrug resistance, that this can basically be conferred by only one or two naturally occurring genetic recombination events. It turns out that virulence factors and drug resistance genes naturally "travel in packs". They are usually found together on little natural miniature chromosomes called plasmids that are easily transferred from bacteria to bacteria, or in whole regions of the main chromosome (or plasmids) called "pathogenicity islands" that were transferred as a group by viruses that infect bacteria. This happens all the time... Turn your back on some bacteria for a moment and they'll drop their pants and start swapping spit and recombining their genes faster than you can say kiss my hand. Moreover, you really don't need that many different resistance genes to confer resistance to a number of different types of antibiotics... to begin with many of the families are chemically fairly closely related (i.e. "-cillins" and "cephalo-s" both have beta lactam rings). And even when they are not, some antibiotic resistance genes work as (for instance) "efflux pumps" which just pump uninvited molecules out of the bacterium in a fairly non-specific way. I should point out that the antibiotic resistance is not even really the interesting part of this story... it's actually kind of a side-show, by virtue of the fact that prescription of antibiotics is contraindicated in treatment of these conditions... even if they kill the bacteria perfectly successfully, it's been discovered that such treatment just pisses off the bacteria and make them pump out a shitload more toxin before they die. So really, the interesting part of this story has little to do with the antibiotic resistance, which has unfortunately become a relatively unremarkable observation, but rather on the fact that what is basically an enterohemorrhagic strain has acquired some enteroaggregative properties that seem to really kickstart its virulence. In fact, it's a testament to the power of the internet how quickly people have been able to pick apart the whole story of the phylogeny and pathogenicity of this beast. Basically, the analysis of the genetic evidence has been extensively "crowdsourced", and people all over the world have had a chance to analyze the raw sequence data from at least four or five different isolates of the outbreak bug. If you're interested in following one of the more factual blogs documenting this crowdsourced analysis of tbe data, I can recommend this one: http://bacpathgenomics.wordpress.com/ The next question's whether the genes for the toxin occur naturally in the bacteriophage & where they may have picked them up. This is an exceedingly common and well-described phenomenon in bacterial virulence. In fact, a lot of pathogens have been known for a long, long time to depend on bacteriophage for carrying their virulence genes. Just to name a few famous examples: Vibrio cholera (cholera) - the CTX phage carries the cholera toxin; Streptococcus pyogenes (Scarlet fever) - the T12 phage carries the toxin genes; Corynebacterium diphtheriae (diptheria) - phage Beta carries the diptheria toxin... shall I continue? Oh, yeah - lambdoid phages of E. coli often carry shigatoxin-like toxins. So there you go. If you're interested in finding out more, google the term "Lysogenic conversion".. It should start you down an interesting path.

hype to avoid like the plague

Submitted by tentontoby on Fri, 09/30/2011 - 10:48.
There are also no scientifically reputable sources for the claim that there are Y. pestis specific virulence factors in O104:H4

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